Alternative Medicine

             The Cancer Cure they don’t want you to have!

             Here I will show mountains of medical references as proof…

             Proof they ignore, and lie about!  (Print this for your study.)

One of the most common nitrilosides is amygdalin. This nitriloside occurs in the kernels of seeds of practically all fruits. The seeds of apples, apricots, cherries, peaches, plums, nectarines, and the like carry this factor; often in the extraordinary concentration of 2 to 3 per cent. Since the seeds of fruits are possibly edible, it may be proper to designate the non-toxic water soluble accessory food factor or nitriloside that they contain as vitamin B-17.

The presence of nitriloside in the diet produces specific physiologic effects and leaves as metabolites specific chemical compounds of a physiologically active nature. The production by a non-toxic, water-soluble accessory food factor of specific physiological effects as well as identifiable metabolites suggests the vitamin nature of the compound.

In metabolism, nitnloside is hydrolyzed to free hydrogen cyanide, benzaldehyde or acetone and sugar. This occurs largely through the enzyme Beta-glucosidase produced by intestinal bacteria as well as by the body. The released HCN [hydrocyanide] is detoxified by the enzyme rhodanese to the relatively non-toxic thiocyanate molecule. The sugar is normally metabolized. The released benzaldehyde in the presence of oxygen is immediately oxidized to benzoic acid which is non-toxic. Thus this newly designated vitamin B-17 (nitriloside) could account for:

1. The thiocyanates in the body fluids—blood, urine, saliva, sweat, and tears;
2. For part of the benzoic acid (and subsequently hippuric acid); salicylic acid isomers;
3. For the HCN that goes to the production of cyanocobalamin from hydrocobalamin, or production of vitamin B12 from provitamin B12.

These are the physiological properties of the common mtriloside amygdalin. Before considering the possible antineoplastic activity of this vitamin B-il, let us recall that the benzoic acid arising from it has certain antirheumatic and antiseptic properties. It was rather widely used (in Gennany and elsewhere) for rheumatic disease therapy prior to the advent of the ortho-hydroxy addition product of benzoic acid known as ortho-hydroxybenzoic acid or salicylic acid. It was originally obtained from beech-wood bark. As a matter of internet, the pam- hydroxy isomer of benzoic acid occurs in the para hydroxybenzaidehyde aglycon (non-sugar) of the nitnloside found in the cereal millet. Millet was once more widely used in human nutrition than wheat. Wheat seed contains little or no nitriloside.

Recall now, that thiocyanate also was once widely used, in both Germany and American medicine, as an effective agent for hypertension. Used as such, as the simple chemical, the dosage was difficult to control. Obviously, this difficulty does not arise from the thiocyanate usually produced in the body through metabolizing vitamin B-Il (nitriloside). However, chronic hypotension has been reported in Nigerians who eat quantities of the mtriloside-containing mamoc (cassava)—especially that of the bitter variety. (Also eaten in the Philippines.)

Let us pause to reflect upon this question: Might not the rheumatic diseases as well as certain aspects of hypertension be in some cases partially related to a dietary deficiency in mtrilosides? One can hardly deny that the ingestion of a sufficient quantity of nitriloside-containing foods will metabolically yield sufficient benzoic acid and/or salicylic acid isomers to palliate rheumatic disease and certainly to decrease, however temporarily, hypertension as well as to foster the mtrilosation of provitamin B-12 to active vitamin B-i2: cyanocobalamin.

Despite all this, are we justified in suggesting that cancer itself might be another chronic metabolic disease that arises from a specific vitamin deficiency—a deficiency specifically in vitamin B-il (nitriloside)?

There are many chrome or metabolic diseases that challenge medicine. Many of these diseases have  One of the most common nitrilosides is amygdalin. This mtriloside occurs in the kernels of seeds of practically all fruits. The seeds of apples, apricots, cherries, peaches, plums, nectarines, and the like carry this factor; often in the extraordinary concentration of 2 to 3 per cent. Since the seeds of fruits are possibly edible, it may be proper to designate the non-toxic water soluble accessory food factor or nitriloside that they contain as vitamin B-17.

THE BACKGROUND ON WHY THE FDA FORBIDS ITS USE Amygdalin
AMYGDALIN (Vitamin B-17)

                                            By Dr. Manuel D. Navarro M.D.
                                          NUTRITIONAL ONCOLOGIST

(This article was written by Dr. Navarro several years before he passed away in 1994.)

     “The American FDA claims that Vitamin B17 is poisonous in its propaganda – this herbal medicine obtained from over 1,200 species of plants distributed all over the world.”

     “The FDA lied in its propaganda poster wherein it stated that B17 contains cyanide which is half true. What it contains is the cyanide radical (CN) which needs to have an H ion before it becomes poisonous. The molecule of Vitamin B17 is in itself not poisonous until it is hydrolyzed or split by an enzyme called Beta glucosidase or glucuronidase (B.G). when B17 is injected into a cancerous patient, the B.G. around the cancer cells splits the B17 and the HCN produced diffuses into the cancer cells poisoning them, while the normal cells nearby are not poisoned due to the presence of an enzyme, rhodanese, - not found in the cancer cells. The rhodanese adds an Atom of Sulfur to the CN to form SCN, a very mild hypotensive.”

     “It was brought out in the American Court trying a doctor who used B17 for his cancerous patient that the FDA has a large list of accepted drugs produced in the United States that are generally regarded as safe (GRAS list). This Gras list contains Amygladin and Amygladin is synonymous with Laetrile, the trade name of Vitamin B17.

Since then the FDA in its shame has stopped using this ploy to downgrade the use of Vitamin B17 that is highly competitive with the various drug products of the drug cartels approved by the FDA although they are more toxic than SCN produced from B17. The reactions of the FDA approved drugs are vomiting, loss of appetite, lowering of blood count suppression of the bone marrow where the immune body defenses are formed with the red and white cells and platelets to the detriment of the patients. The approved drugs poison the cancer cells as well as the normal cells!”

     “Orally, Vitamin B17 in the correct dosage of 2-4 grams a day for adult, in divided doses is very well tolerated. The intravenous administration of higher dosages does not produce any reaction. A baby girl of 4 months suffering from cancer of the adrenal that has spread to the liver, bones, etc, received Vitamin B17 per rectum and she got well her ailment. She celebrated her third birthday recently.”

      “The Mayo Clinic in the issue of the American Journal of the America Medical Association of February 13, 1981 declared Vitamin B17 is safe, orally or parentally in the correct dosages. In the 36 years of using it we have not met yet with any toxic reaction.”

 (This article above about the FDA was given to me as a paper written by Dr. Navarro in 1989.)

Dr. Navarro was my doctor from 1998 until he died in 1994 He had me in remission from my fist size colon cancer in three weeks using B-17, diet, vitamins and digestive enzymes. And was fine under his Alternative care. However two years after he passed away, I was in trouble again, infected with intestinal worms. I made my invention (seen on <www,geneticenergy.org> and it not only killed my worms bur finished off my cancer too!

The presence of nitriloside in the diet produces specific physiologic effects and leaves as metabolites specific chemical compounds of a physiologically active nature. The production by a non-toxic, water-soluble accessory food factor of specific physiological effects as well as identifiable metabolites suggests the vitamin nature of the compound.

In metabolism, nitriloside is hydrolyzed to free hydrogen cyanide, benzaldehyde or acetone and sugar. This occurs largely through the enzyme Beta-glucosidase produced by intestinal bacteria as well as by the body. The released HCN [hydrocyanide] is detoxified by the enzyme rhodanese to the relatively non-toxic thiocyanate molecule. The sugar is normally metabolized. The released benzaldehyde in the presence of oxygen is immediately oxidized to benzoic acid which is non-toxic. Thus this newly designated vitamin B-17 (nitriloside) could account for:

1. The thiocyanates in the body fluids—blood, urine, saliva, sweat, and tears;
2. For part of the benzoic acid (and subsequently hippuric acid); salicylic acid isomers;
3. For the HCN that goes to the production of cyanocobalamin from hydrocobalamin, or production of vitamin B12 from provitamin B12.

These are the physiological properties of the common mtriloside amygdalin. Before considering the possible antineoplastic activity of this vitamin B-il, let us recall that the benzoic acid arising from it has certain antirheumatic and antiseptic properties. It was rather widely used (in Gennany and elsewhere) for rheumatic disease therapy prior to the advent of the ortho-hydroxy addition product of benzoic acid known as ortho-hydroxybenzoic acid or salicylic acid. It was originally obtained from beech-wood bark. As a matter of interet, the pam- hydroxy isomer of benzoic acid occurs in the para hydroxybenzaidehyde aglycon (non-sugar) of the nitnloside found in the cereal millet. Millet was once more widely used in human nutrition than wheat. Wheat seed contains little or no nitriloside.

Let us pause to reflect upon this question: Might not the rheumatic diseases as well as certain aspects of hypertension be in some cases partially related to a dietary deficiency in mtrilosides? One can hardly deny that the ingestion of a sufficient quantity of nitriloside-containing foods will metabolically yield sufficient benzoic acid and/or salicylic acid isomers to palliate rheumatic disease and certainly to decrease, however temporarily, hypertension as well as to foster the mtrilosation of provitamin B-12 to active vitamin B-i2: cyanocobalamin.

Despite all this, are we justified in suggesting that cancer itself might be another chronic metabolic disease that arises from a specific vitamin deficiency—a deficiency specifically in vitamin B-il (nitriloside)?

There are many chrome or metabolic diseases that challenge medicine. Many of these diseases haveiseases haveDelga, 3., Mizoula, J., Veverka, B., and Bon, R., Studies on the Treatment of Cyamde Intoxication by

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By Rev. Dr. Howard E. May
Sept. 8, 2007
msgr@Godgift.org

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